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2015). These reports, taken with each other, propose that, While a direct activation of GPR55 or PPAR‐α takes place, PEA can make its anti‐inflammatory motion from the gut also by using indirect activation of CB1 and CB2 receptors, likely as a result of the flexibility of this compound to potentiate the action of endocannabinoids at these rece
Prevalence of Key end result modifications in clinical trials registered on ClinicalTrials.gov: a cross‐sectional research. F1000Res1996). Oral PEA also diminished paw oedema induced by carrageenan, dextran and formalin, suggesting which the compound immediately down‐modulates mast cell activation in vivoIn these studies, PEA-Q has long been sh